Tuesday, August 19, 2025

12-Year study reveals the hidden link between meal timing, genetics and liver health

12-Year study reveals the hidden link between meal timing, genetics and liver health

By Ava Grace

Post on August 19, 2025


A 12-year Spanish study reveals that when you eat is just as crucial as what you eat, especially for those genetically predisposed to obesity. Late eating activates obesity-related genes, while early meals can counteract them.

High-risk individuals who eat late see a significant BMI increase — over two points per hour of delayed eating — while early eaters maintain weights similar to low-risk peers. Late eaters also struggle more with weight loss and maintenance.

Late-night meals disrupt the liver’s natural cycle (nutrient processing by day, detoxification by night), leading to fat storage and increasing the risk of fatty liver disease, now affecting 25 percent of adults.

Those with high genetic risk experience accelerated fat storage and suppressed fat-burning when eating late, while others may temporarily avoid consequences — though rising liver disease rates threaten everyone.

Syncing meals with biology — front-loading calories by mid-afternoon, finishing meals 12 hours before breakfast and prioritizing morning light — can mute genetic risks and support liver health without costly interventions.

A groundbreaking 12-year study from Spain has uncovered a startling truth: When you eat may be just as critical as what you eat — especially if obesity runs in your family. Researchers at Complutense University of Madrid tracked nearly 1,200 adults and found that late eating activates genetic obesity risks while early meals can override them. But the most alarming revelation? The hidden toll of late-night meals on the liver, an organ already under siege by today’s fast food-heavy diets.

The genetic time bomb in your diet

For years, weight-loss advice has focused on calories and exercise, but this study shifts the conversation to timing. Scientists calculated genetic obesity risk scores using nearly a million genetic markers. The results were striking: Those with high genetic risk who ate early maintained weights similar to low-risk individuals. But delay meals, and the scale tips dramatically — every hour of later eating increased BMI by over two points in high-risk individuals.

That difference isn’t trivial — it’s the gap between overweight and obese, dictated not by gluttony but by the clock. Worse, late eaters lost weight slower during treatment and struggled to keep it off long-term.

Your liver’s silent crisis

While the study focused on weight, the liver’s role is equally dire. The liver operates on a strict schedule: by day, it processes nutrients; by night, it detoxifies and burns fat. Late-night meals hijack this rhythm, forcing the liver to store fat instead of cleansing the body. Over time, this leads to fatty liver disease — a condition now affecting 25 percent of adults, with rates climbing alongside modern eating habits.

Why some people pay a higher price

Not everyone suffers equally. Genetics determine sensitivity: High-risk individuals see obesity genes “switch on” with late eating, accelerating fat storage and suppressing fat-burning mechanisms. Their livers, already genetically predisposed to sluggish metabolism, buckle under the strain. Meanwhile, those with favorable genetics might dodge consequences — for now. But as liver disease becomes epidemic, even the resilient may not stay immune.

How to outsmart your genes

The solution isn’t another fad diet — it’s syncing with biology. Front-loading calories by eating half your daily food by mid-afternoon can make a significant difference. Closing the kitchen early — finishing meals 12 hours before breakfast — helps reset the body’s internal clock. Prioritizing morning light exposure aids the liver’s natural rhythm, while liver-friendly foods like cruciferous vegetables, green tea and turmeric support detoxification.

A wake-up call for modern health

This research dismantles the myth that obesity is purely about willpower or calories. It’s a dance between genes and timing — one that modern life disrupts with late work dinners, midnight snacks and erratic schedules. For those genetically vulnerable, the stakes are liver damage, metabolic chaos and futile weight-loss battles.

But there’s hope. Unlike genes, meal timing is controllable. By eating earlier, even high-risk individuals can mute genetic obesity triggers and protect their liver. In an era of rising metabolic disorders, this study offers a simple, no-cost fix: Listen to the clock.

The Spanish study isn’t just another diet headline — it’s a paradigm shift. As science exposes the link between meal timing, genetics and liver health, the message is clear: Supper at sunset might be wiser than midnight feasts. For a nation battling obesity and fatty liver disease, the ancient adage “eat breakfast like a king, dinner like a pauper” may hold the key to survival.

Ava Grace

Monday, August 18, 2025

Understanding Your Thyroid and Autoimmunity


Understanding Your Thyroid and Autoimmunity

By Stephanie Walsh

Posted on August 17, 2025



Your thyroid gland is a tiny butterfly-shaped organ located at the base of your neck near the Adam’s apple. Despite its tiny size, the thyroid has a big job controlling energy production for every cell in the body. The thyroid’s main role is regulating metabolism through two hormones, thyroxine (T4) and triiodothyronine (T3). T4 is an inactive form which gets converted into T3 in the liver, kidneys, gastrointestinal tract, brain and muscles. It is the active form of T3 that stimulates cells to produce energy. The thyroid works in concert with the hypothalamus and pituitary gland to regulate thyroid hormone production. But, the health of your thyroid is also intricately tied to the health of your gastrointestinal tract, gut microbiome, liver, kidneys, adrenal glands and even your immune system.

Problems with thyroid function arise when something throws off the delicate balance between any of these connections. Eventually thyroid hormone production gets out of whack, conversion of thyroid hormone into its active form falters, and/or thyroid tissue gets damaged. Compromised thyroid health may set the stage for a more debilitating autoimmune condition of the thyroid called Hashimoto’s.

What is Hashimoto’s
Hashimoto’s Thyroiditis, commonly known as just Hashimoto’s, is an autoimmune disease in which the body’s immune system attacks the thyroid. This attack gradually destroys the thyroid tissue and its ability to produce critical hormones. According to the American Autoimmune Related Diseases Association, Inc., approximately 20% of the U.S. population suffers from autoimmune diseases and Hashimoto’s is the most common. Even more shocking, thyroid diseases occur roughly five times more frequently in women than men. With such prevalence, it’s very important to understand its causes and effects as well as how you can effectively prevent or treat this common condition.

Causes and Risk Factors
As an autoimmune condition, Hashimoto’s arises from a dysfunctional immune system, not a dysfunctional thyroid. This means the condition is actually a symptom of a much deeper issue. Stress on the thyroid and/or immune system are at the root of Hashimoto’s and there are many stressors at play.

The first thing to consider is an overactive immune system. The primary cause for an overcharged immune system is inflammation. If the inflammatory process never gets a rest, it stays in overdrive. This can occur because of:

  • Infections, both chronic and acute

  • Injuries

  • Overload of toxins

  • Diet lacking in antioxidants

  • Intestinal permeability or “leaky gut”

Additionally, there are other potential causes for Hashimoto’s:

  • Exposure to radiation, through work or medical treatments.

  • Excess exposure to environmental toxins, fluoride and perchlorate in water, mercury and other heavy metals, lithium, and estrogens from pesticides and hormone creams or pills.

  • Overconsumption, or deficiency, of dietary iodine.

  • Overconsumption of soy products and uncooked “goitrogenic” foods such as broccoli, Brussels sprouts, cabbage, cassava, cauliflower, kale, kohlrabi, millet, radishes, rutabaga and turnip.

  • Leaky gut syndrome, food allergies, gut dysbiosis, nutrient deficiencies and poor digestive health.

  • Chronic blood sugar imbalance, insulin resistance and diabetes.

  • Chronic inflammation.

  • Hormonal imbalances.

  • Liver, pituitary, hypothalamus and/or adrenal dysfunction.

  • Inappropriate use of thyroid medications.

  • Presence of other autoimmune conditions such as Type 1 diabetes, celiac disease, lupus, rheumatoid arthritis, and/or multiple sclerosis.

  • Chronic stress.

The major risk factors are chronic stress, gluten intolerance or celiac disease, insulin resistance, vitamin D deficiency, leaky gut and compromised detoxification. All of these risk factors create a vicious cycle of continually increasing stress on the body. Chronic and prolonged stress then increases damage to the adrenals, gut, liver and pituitary as well as compromises blood sugar, nutrient absorption and immunity. The cycle continues until the immune system spirals out of control creating chronic inflammation and an overactive immune system, the perfect storm leading to Hashimoto’s.

Gut Health
A leaky gut is not just a source of inflammation, it is a cog in a dangerous cycle that spirals out of control. The cycle starts with a disruption in the gut lining from food intolerances, alcohol, stress, anti-inflammatory drugs, antibiotics, candida overgrowth, or fatty acid deficiency. This leads to malabsorption of nutrients and undigested food particles directly entering the bloodstream which creates an immune response. The immune response creates immune complexes that settle in the gut lining and create inflammation. This inflammation continues to damage the gut lining which leads to food allergies/intolerances, more disruption in the lining, more malabsorption, more inflammation and more leaky gut.

Gut health is very important for immune and thyroid health. Over 70% of your immune system is in the gut, and 20% of circulating T4 is converted to T3 by an enzyme in your gastrointestinal tract. If the gut lining is compromised your immunity weakens and your production of active thyroid hormone decreases.

An underlying gluten intolerance may be the most damaging risk factor. Thyroid tissue and the gluten molecule have a similar protein structure. If the body mounts an attack on gluten, it may also inadvertently attack thyroid tissue. It is a trifecta of autoimmune risk, a compromised gut, an overactive immune system, and a diet high in gluten. Your body attacks gluten while also mistakenly attacking healthy thyroid tissue. This is the very definition of Hashimoto’s Thyroiditis. In fact, Hashimoto’s is always associated with gluten sensitivity. If you suspect Hashimoto’s, already have an autoimmune condition, or suffer from a compromised immune system you should completely avoid gluten to protect your thyroid.

The Varied Symptoms
Many things can increase your risk of Hashimoto’s. It is also common to have many symptoms, including:

  • Fatigue

  • Excessive sleep

  • Depression

  • Poor concentration and memory

  • Weight gain

  • Dry and brittle hair and nails

  • Thinning eyebrows

  • Muscle and joint pain

  • Morning headaches

  • Tendonitis

  • Puffy eyes and face

  • Cold hands and feet

  • Full sensation in neck

  • Raspy, hoarse voice

  • Heart palpitations

  • Insomnia

  • Shortness of breath

  • Low sex drive

  • Infertility, heavy periods and PMS

  • High cholesterol

  • High blood pressure

  • Insulin resistance

  • Worsening allergies

  • Chronic infections, including yeast and sinus infections

  • Slow wound healing

  • Heartburn and difficulty swallowing

  • Indigestion, nausea, gas and bloating

  • Constipation and other digestive issues

Diagnosing Hashimoto’s
The most common diagnostic test for thyroid dysfunction is testing levels of thyroid stimulating hormone (TSH). However, to confirm Hashimoto’s it is important to test for thyroid peroxidase antibodies (TPO Ab) and thyroglobulin antibodies (TGB Ab). A full Thyroid Panel which includes Total T4, Free T4 Index, Free T4, Free T3, T3 Uptake, Reverse T3 and Thyroid Antibodies will be beneficial to determine the source of dysfunction and the degree of damage. If you are diagnosed with Hashimoto’s, you have both conventional and alternative treatments to consider.

Treatment Options
Conventional treatment for Hashimoto’s is to monitor thyroid hormone levels, adjust medications accordingly, and prescribe more medications to suppress any symptoms. This could include thyroid hormone as well as Prozac for depression, or medications to slow heart rate or adrenal function. Treatment may even lead to partial or complete removal of the thyroid and a lifetime of hormone replacement therapy. The concern with conventional treatment, however, is that it does not address the underlying issue, an overactive immune system and the root causes of its hyperactivity.

Alternative and holistic minded treatments address the root cause of an overactive immune system. Alternative medicine practitioners will determine the health of your gastrointestinal tract, liver and adrenal glands; your level of exposure to toxins and excess estrogen; as well as the quality of your current diet and how it may contribute to nutrient deficiencies, toxin exposure, blood sugar imbalance and stress on the body. Also, through blood tests they can determine not only current thyroid function, but what may be at the root cause. Through this, they can determine the course of treatment. In any scenario, regulating the immune response with targeted nutrition and supplementation will be critical.

Lifestyle Changes for Hashimoto’s
If you have been diagnosed with Hashimoto’s, hypo- or hyperthyroidism, or any other autoimmune disease, the most important dietary change is to completely remove gluten from your diet. Consuming gluten when the immune system is already overactive will actually increase damage and destruction of the thyroid.

Here are lifestyle changes that can reduce your risk, or improve your symptoms, of Hashimoto’s:

  • Test for other food allergies.

  • Consume a diet high in healthy fats, fiber, clean protein and organic produce, and low in carbohydrates to help balance blood sugar, increase immunity and support the thyroid.

  • Eat every 2-4 hours.

  • Avoid soy, dairy and sugar as well as gluten.

  • Choose organic to reduce pesticide and toxin exposure.

  • Cook goitrogenic foods such as kale, cabbage, turnips, sweet potatoes, kelp, broccoli and Brussel sprouts. Goitrogen is a compound that inhibits iodine uptake by the thyroid. Lightly cooking these veggies will prevent this negative effect.

  • Consider a safe liver detox protocol.

  • Practice stress management techniques daily.

  • Incorporate a mix of short cardio sessions and strength training for exercise.

  • Avoid iodine supplementation if you are diagnosed with Hashimoto’s, it can further damage the thyroid.

Supplements for Hashimoto’s
Certain supplements may also help support the thyroid:

  • Selenium may reduce inflammation, assist with conversion to T3 and protect thyroid function.

  • Magnesium may help balance blood sugar and support the pituitary-thyroid connection.

  • Vitamin C may enhance immunity, repair the gut lining, and reduce inflammation.

  • Vitamin D is key for regulating the immune system.

  • B Vitamins may support proper digestion, immunity and thyroid function.

  • Zinc may promote thyroid hormone production and enhance immunity.

  • Maca Root may enhance thyroid function.

  • Milk Thistle may support detoxification and reduce inflammation.

Remember, your body systems and organs are all intimately connected. Focusing on thyroid health will naturally enhance your overall health. The more you support your thyroid with quality nutrition and targeted supplementation, the more you can calm your immune system, heal your gut and optimize your health.

Stephanie Walsh

Saturday, August 16, 2025

Breaking the Link Between Alcohol Use and Pancreatic Cancer


Breaking the Link Between Alcohol Use and Pancreatic Cancer

By Charlotte Schubert

Post on August 16, 2025


The U.S. surgeon general recently declared alcohol the third-leading preventable cause of cancer. Yet, despite increasing evidence linking alcohol consumption to the disease, little is known about the biological mechanisms behind the association.

new study by researchers at Sylvester Comprehensive Cancer Center, part of the University of Miami Miller School of Medicine, provides insight into these mechanisms in pancreatic cancer.

The new findings delved into the role of a cellular molecule called CREB. CREB seems to mediate an inflammatory condition in the pancreas caused by high alcohol use that increases the risk of developing cancer. Inhibiting CREB might thwart pancreatic tumor development in response to alcohol.

“Our model serves as an important platform for understanding how chronic inflammation related to alcohol consumption accelerates the development of pancreatic cancer,” said Sylvester scientist Siddharth Mehra, Ph.D., who is the study’s first author.

The Alcohol-Cancer Connection

Chronic, high alcohol use damages the acinar cells in the pancreas that produce digestive enzymes. The damage in turn causes the cells’ enzymes to increase inflammation in the tissue, exacerbating damage to the pancreas.

Over time, precancerous lesions can develop, increasing the risk for full-blown pancreatic cancer, one of the deadliest types of tumors. Previous studies have implicated CREB, a DNA-binding protein that regulates gene activity, and associated molecules in helping to mediate this process.

Progression to cancer also generally requires other cellular events, such as a mutation in a pro-cancerous gene called Ras, which commonly occurs in pancreatic tumors.

In the new study, the researchers developed a model that recapitulated alcohol-induced inflammation, the development of pre-cancerous lesions and progression to cancer. The model contained Ras mutations in acinar cells, and it also had an intact CREB gene that could be experimentally knocked out in these cells.

A Molecular Orchestrator

The researchers found that exposure to alcohol and a pro-inflammatory molecule caused the development of symptoms similar to alcohol-induced pancreatitis, an inflammatory condition. Inflammation in turn prompted the development of precancerous lesions and, later, cancer. Consistent with previous studies, CREB was highly activated throughout this transition process.

The researchers next knocked out CREB and found they could quell the development of precancerous and cancerous lesions, even in the continued presence of alcohol. Knocking out CREB also relieved damage to acinar cells.

We believe this study lays the groundwork for future translational efforts targeting CREB as a therapeutic vulnerability in inflammation-associated pancreatic cancer.

Dr. Nipun Merchant

The findings hint that inhibitors of CREB might have therapeutic potential in people who have high alcohol use. Such inhibitors could potentially relieve damage to the pancreas and thwart tumor development, said the researchers.

“We found that CREB is not just a mediator of inflammation. It is a molecular orchestrator that permanently converts acinar cells into precancerous cells, which ultimately progress to high-grade neoplasia,” said senior author Nagaraj Nagathihalli, Ph.D., associate professor in the DeWitt Daughtry Family Department of Surgery and assistant director of the Sylvester Pancreatic Cancer Research Institute at the University of Miami.

The findings appeared Aug. 12 in the journal, Cellular and Molecular Gastroenterology and Hepatology. The research was funded in part by the U.S. National Institutes of Health as part of an effort to probe the biology behind alcohol use and cancer.

Setting the Stage

Future studies should help provide additional information about how alcohol use promotes pancreatic cancer development.

Points of investigation include whether similar events occur in human cells and tissues and what other molecules and cells play a role in the process. CREB activation may also be involved in other alcohol-linked cancers.

Dr. Nagathihalli and his colleagues are also leveraging the model to investigate the potential of CREB inhibitors, which are under development as potential cancer therapeutics.

“We believe this study lays the groundwork for future translational efforts targeting CREB as a therapeutic vulnerability in inflammation-associated pancreatic cancer,” said study co-author Nipun Merchant, M.D., Sylvester associate director of translational science and chief of the Division of Surgical Oncology at the Miller School.

Charlotte Schubert

Thursday, August 14, 2025

The 10 Best Foods to Boost Nitric Oxide Levels


The 10 Best Foods to Boost Nitric Oxide Levels

By Rachael Ajmera

Post on August 14, 2025


Nitric oxide has important roles in your body, such as helping to lower blood pressure, improve blood flow, and boost exercise performance. To increase nitric oxide levels, you can eat foods such as beets, garlic, meat, leafy greens, and citrus.
Nitric oxide is a vital molecule produced in your body that affects many aspects of health.

It helps your blood vessels dilate to promote proper blood flow and may provide various other health benefits, including improved exercise performance, lower blood pressure, and better brain function.

Switching up your diet is one of the best and most effective ways to naturally boost your levels of this important molecule.

Here are the 10 best foods to boost your nitric oxide levels.

1. Beets

Beets are rich in dietary nitrates, which your body can convert to nitric oxide.

In one small study in 38 adultsTrusted Source, consuming a beet juice supplement increased nitric oxide levels by 21% after just 45 minutes.

Similarly, a 2016 studyTrusted Source showed that drinking 3.4 ounces (oz), or 100 milliliters (mL), of beet juice significantly increased nitric oxide levels in both men and women.

Thanks to their rich content of dietary nitrates, beets have been linked to a number of health benefits, including improved thinking ability, enhanced athletic performance, and lower blood pressure levels.

2. Garlic

Garlic can boost nitric oxide levels by activating nitric oxide synthase, the enzyme that helps convert the amino acid L-arginine to nitric oxide.

One 2015 animal studyTrusted Source showed that aged garlic extract helped lower elevated levels of lipids such as cholesterol and triglycerides. But it’s important to note that using aged garlic extract or garlic oil is not the same as eating garlic.

2013 test-tube study also suggests that aged garlic extract can help maximize the amount of nitric oxide that the body can absorb.

Both human and animal studies indicate that garlic’s ability to increase nitric oxide levels may have health benefits and can help lower blood pressureTrusted Source and improve exercise toleranceTrusted Source.

3. Meat

Meat, poultry, and seafood are all excellent sources of coenzyme Q10 (CoQ10), an important compound that is believed to help preserve nitric oxide in the body.

Organ meats, fatty fish, and muscle meats such as beef, chicken and pork contain the highest concentration of CoQ10.

Studies show that getting enough CoQ10 in your diet not only preserves nitric oxide but also can help improveTrusted Source athletic performance, preventTrusted Source migraine headaches, and promoteTrusted Source heart health.

4. Dark chocolate

Dark chocolate is loaded with flavanols, naturally occurring compounds that boast an extensive list of powerful health benefits.

In particular, researchTrusted Source shows that the flavanols found in cocoa can help establish optimal nitric oxide levels in your body to promote heart health and protect cells against oxidative damage.

One older 15-day studyTrusted Source in 16 people showed that consuming 30 grams of dark chocolate daily led to significant increases in blood levels of nitric oxide.

What’s more, participants experienced decreases in both systolic and diastolic blood pressure (the top and bottom numbers of a blood pressure reading, respectively).

Because of its rich content of nitric oxide-boosting flavanols, dark chocolate has been associatedTrusted Source with improved blood flow and lower blood pressure.

5. Leafy greens

Leafy green vegetables such as spinach, arugula, kale, and cabbage are packed with nitrates, which your body converts to nitric oxide.

Regular consumption of nitrate-rich foods like green leafy vegetables can help maintain sufficient levels of nitric oxide in your blood and tissues.

One 2020 study even showed that eating a nitrate-rich meal containing leafy greens or beet juice equally increased nitrate levels and significantly decreased both systolic and diastolic blood pressure.

Other researchTrusted Source has foundTrusted Source that consuming high nitrate leafy greens may also be associated with a reduced risk of heart disease and cognitive (mental) decline.

6. Citrus fruits

Citrus fruits such as oranges, lemons, limes, and grapefruit are all excellent sources of vitamin C, an important water-soluble vitamin that plays a central role in health.

Vitamin C can enhance levels of nitric oxide by increasing its bioavailability and maximizing its absorption in your body.

Vitamin C may also bump up levels of nitric oxide synthase, the enzyme needed for nitric oxide production.

Studies indicate that citrus fruit consumption may be linked to improved brain functionTrusted Source and decreased risk of cardiovascular problemsTrusted Source. These effects may be due in part to these fruits’ ability to boost nitric oxide levels.

7. Pomegranate

Pomegranate is loaded with potent antioxidants that can protect your cells against damage and preserve nitric oxide.

One older test-tube studyTrusted Source showed that pomegranate juice was effective in protecting nitric oxide from oxidative damage while also increasing its activity.

small 2019 studyTrusted Source in 60 people with type 2 diabetes suggested that pomegranate juice had some benefit for lowering blood pressure, but it didn’t confirm any change in the participants’ lipid profiles.

Recent researchTrusted Source has found that antioxidant-rich pomegranate can improve blood flow, which may be especially beneficial for treating high blood pressure.

In addition, a 2016 animal studyTrusted Source found pomegranate juice to be helpful in treating erectile dysfunction. But more human studies are needed to explore these potential positive effects of pomegranate juice and extract.

8. Nuts and seeds

Nuts and seeds are high in arginine, a type of amino acid that is involved in nitric oxide production.
Some research suggests that including arginine from foods such as nuts and seeds in your diet can help increase nitric oxide levels in your body.

For example, one studyTrusted Source in 2,771 people showed that a higher intake of arginine-rich foods was associated with higher levels of nitric oxide in the blood.
Thanks to their arginine content and stellar nutrient profile, regularly eating nuts and seeds has been associated with lower blood pressure, improved thinking ability, and increased endurance.

9. Watermelon

Watermelon is one of the best sources of citrulline, an amino acid that your body converts first to arginine and then to nitric oxide.

One small 2015 studyTrusted Source found that citrulline supplements helped stimulate nitric oxide production after just a few hours, but the authors noted that it may take longer to see positive effects on health.

A small 2016 study in 8 men showed that drinking 10 oz (300 mL) of watermelon juice for 2 weeks led to significant improvements in nitric oxide bioavailability.

According to a 2017 reviewTrusted Source, upping your intake of watermelon not only enhances nitric oxide levels but also can improve exercise performance, decrease blood pressure, and boost blood flow.

However, a 2022 reviewTrusted Source suggests that citrulline supplements may be a more effective way of obtaining these benefits, since eating watermelon every day may not be practical for everyone.

10. Red wine

Red wine contains many powerful antioxidants and has been linked to some health benefits.

Interestingly, some studies have found that drinking red wine could also increase nitric oxide levels.

2016 reviewTrusted Source of studies found that resveratrol in red wine is likely to provide benefits against cardiovascular disease by improving the availability of nitric oxide, but more human clinical trials are needed.

For this reason, it’s not surprising that moderate consumption of red wine has been thought to reduce blood pressure and improve heart health.
The bottom line

Nitric oxide is a crucial compound involved in many aspects of health, including blood pressure regulation, athletic performance, and brain function.

Making a few simple swaps in your diet can be an easy and effective way to increase your levels of nitric oxide naturally.

Eating plenty of fruits, vegetables, nuts, seeds, and healthy protein foods can help you optimize your nitric oxide levels while promoting better overall health.

Rachael Ajmera